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Riboflavin, in the form of flavin adenine dinucleotide (FAD), is shown as the required cofactor for this reaction, which consumes NADPH and a proton (H + ) and releases NADP +

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Imbalance of Homocysteine and H2S: Significance, Mechanisms, and Therapeutic Promise in Vascular Injury. Oxidative Medicine and Cellular Longevity, vol

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