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To determine if GSH precursors accumulate and are possibly rerouted upon halting GSH synthesis in vivo, we performed targeted metabolite analysis on tissues from Gclc WT and KO mice

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Ectopic overexpression of NDUFS8, achieved through a lentiviral construct, demonstrated a pro-angiogenic impact by enhancing cell proliferation, migration, and capillary tube formation in HUVECs and other endothelial cell types