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And dismissing emerging science simply because its new isnt evidence-based either

3.3 Oxidative stress and mitochondrial dysfunction EED-induced oxidative stress represents a central mechanism underlying reproductive toxicity, with mitochondrial dysfunction serving as both cause and consequence of excessive reactive oxygen species production

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Early optimisation of glycaemic control is key to reducing the incidence of microvascular and macrovascular complications as demonstrated by the landmark DCCT [6]

Possible sediment should not be deemed a fault

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