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Upon ligand binding, GLP-1R activation enhances glucose-dependent insulin secretion, suppresses glucagon release, delays gastric emptying, and reduces appetite, collectively improving glycemic control and promoting clinically meaningful weight reduction [19]
HR for GLP-1-receptor agonists 1.11 [95% CI 0.54, 2.25]), incretin-based drugs can be used as lifelong treatments and studies with longer follow-up time would be needed when such data are available
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