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These findings suggest that the inhibition of EBV reactivation by TNF- is achieved by its primary receptor, TNFR1
Practical and Administrative Reasons Resource Management: GP surgeries operate on tight schedules and budgets
GPx1 and the mechanisms of cancer susceptibility B
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Several lines of evidence point to upregulation of vascular endothelial growth factor receptor 2 (VEGFR2) signaling and modulation of the nitric oxide system [Huang et al., Vascular Pharmacology, 2015]
A case for change
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