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Cognitive function and neuroprotection Selanks nootropic potential has been the subject of numerous rodent studies

Manifestations of oxidative stress have also been reported in the hippocampus and temporal cortex of ASD patients [119, 120]

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While mouse models are invaluable for their low cost, rapid breeding cycle, and genetic tractability, their primary limitation is the failure to spontaneously develop human-like lung disease, which is the main cause of morbidity in patients

Cagrilintide mimics amylin, a different hormone, and acts on amylin/calcitonin receptors

Barberine has also shown neuroprotective effects via elevating the mitochondrial membrane potential (MMP) (Moneim, 2015)