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It has multiple actions including: potentiation of glucose-mediated insulin secretion mechanisms identified: increased -cell proliferation, resulting in an increase -cell mass (Fusco et al, 2017) stimulation of insulin biosynthesis at the translational level, helping to maintain -cell insulin stores and secretory capacity (Baggio & Drucker, 2007) because the GLP-1 effect is glucose-dependent (there is more insulin release when glucose levels are elevated, but less effect when glucose levels are normal), GLP-1 agonists have a lower risk for producing hypoglycemia compared to sulfonylureas (that chronically stimulate insulin release, independent of glucose concentration) suppression of postprandial glucagon release Evidence indicates that stimulation of pancreatic cells by GLP-1 increases their glucose sensitivity, resulting in less glucagon release at any glucose level (Baggio & Drucker, 2007)
Cch dng: Ung 2 vin/ngy, trc hoc sau n
c bit, reduced glutathione c kh nng ti to cc cht chng oxy ha khc nh Vitamin C v Vitamin E sau khi chng b oxy ha, to thnh vng tun hon bo v lin tc
Am J Trop Med Hyg 83(1):171173 Lyu H et al (2020) Characterization of lead compounds targeting the selenoprotein thioredoxin glutathione reductase for treatment of schistosomiasis
Glutathione anabolism is regulated by the transcription factor Nrf2 and is an important antioxidant metabolic pathway
