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2017 Sep;83:2541
Although these findings are provocative, several limitations need to be considered
In conclusion, our data revealed that CCW16 covalently bound to a large variety of different proteins thereby inducing a cell death mechanism based on oxidative stress and ferroptosis in an RNF4 independent manner
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Fosgonimeton has neuroprotective effects in both LPS (HY-D1056) -induced neuroinflammation and A-induced AD models
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