Different hypotheses have been proposed to explain the pathologic process of AD, including the cholinergic hypothesis [17], the tau hypothesis [18, 19], the glutamate dysfunction hypothesis [20], the amyloid cascade hypothesis [21, 22], the inflammatory hypothesis [23], and the mitochondrial cascade hypothesis [24]
Multipoint imprinting analysis indicates a common precursor cell for gonadal and nongonadal pediatric germ cell tumors1
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