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Papaioannou A, Liakopoulou A, Papoulis D, Gianni E, Gkolfi P, Zygouri E

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Known molecular mechanisms driving LEC fibrosis following lens injury/cataract surgery Currently, it is generally accepted that production of fibrotic tissue following lens injury leading to anterior subcapsular cataract, or after cataract surgery to create cells capable of contributing to fibrotic PCO, is driven by transdifferentiation of LECs to myofibroblasts via epithelial to mesenchymal transition (36, 49, 153, 185)

Congenital anomalies and spontaneous abortion in mice resulting from the use of escitalopram