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In tumors, persistent ROS elevation increases GSH synthesis and shifts the redox balance, creating a biochemical environment that selectively activates disulfide/diselenide linkers and metal-based GSH-depleting nanoplatforms

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When this control is changed by oxidative or electrophilic signals, Nrf2 can accumulate and increase the expression of antioxidant-response genes, including those involved in GSH synthesis

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Li et al., 2013), and others (Wang et al., 2013) have been described and praised for their intrinsic peroxidase-like activities in a variety of assays

Voltage-gated sodium channels