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Witschi A, Junker E, Schranz C, Lauterburg BH
found that accumulation of iron and ROS, dysfunction of iron metabolism, decreased GPX4 activity, and mitochondrial atrophy after TBI are all typical characteristics of ferroptosis
However, this study has certain limitations that warrant further investigation
Zhan, T., Rindtorff, N
In this context, targeting QA, or the enzymatic steps that govern the balance of KP metabolites, may represent a more tractable therapeutic strategy, allowing attenuation of NMDAR-driven excitotoxicity without disrupting 7nAChR-dependent cognitive processes
Gulati, N., et al
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