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Results CNS metabolic shifts in neuroinflammation coincide with monocyte influx To identify EAE-triggered metabolites and understand their relationship with myeloid cell influx, we immunized mice with myelin oligodendrocyte glycoprotein peptide 3355 (MOG 3555 ) in complete Freunds adjuvant, collected spinal cord tissue and CSF at peak disease or during recovery, performed targeted metabolomics and subsequently categorized metabolites at homeostasis and during different EAE phases (Fig
In the lab, glutathione peroxidase improved Huntingtons disease
Shahzad, K
Enns and T.M
Curve fitting and bias calculation were performed as described above in the relevant Methods section
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