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Although malonyl-CoA and long-chain acyl-CoA are believed to regulate GSIS, abrogation of malonyl-CoA formation by overproducing malonyl-CoA decarboxylase in beta cells did not interfere with GSIS, [58] whereas chronic, but not acute, suppression of ACC production by 6080% in INS-1 832/13 cells and isolated islets significantly reduced GSIS [59]
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This highlights the precision and rapid distribution achieved through IV administration [7]
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Ethics declarations Ethics approval and consent for publication The present study was approved by the CPRD Research Data Governance (#23_002545), the Research Ethics Board of Sunnybrook Health Sciences Centre (PIN: 6013), and the Research Ethics Board of University of Toronto (#46463)
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