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These cells, which accumulate with age, no longer divide but resist programmed cell death, contributing to inflammation and tissue dysfunction

Unfortunately, this initial phase often converts toxins into highly reactive intermediate metabolites that are actually more dangerous and damaging than the original substance

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Center for Drug Evaluation and Research (CDER) and Center for Biologics Evaluation and Research (CBER) U.S

Here, we show that nonhomologous end-joining employs both of two a priori possible strategies

The result is more side effects with no extra benefit