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Published microarray analyses have reported effects on genes involved in anti-inflammatory responses, antioxidant defense systems, stem cell markers, and DNA repair pathways
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Randomization was conducted by an independent researcher who was not involved in patient enrollment, treatment administration, or outcome evaluation, ensuring allocation concealment
Our data demonstrate that in ILC2s, melatonin suppresses effector function through a receptor-independent metabolic mechanism involving NRF2 activation and glutathione biosynthesis
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