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Rather than acting solely through weight loss or peripheral glucose control, GLP-1 therapies engage neural pathways involved in energy regulation, inflammation, synaptic integrity, and protein homeostasisoffering a biologically coherent explanation for their emerging association with preserved cognitive function and reduced dementia risk
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Findings include: Accelerated wound closure rates in multiple animal models across all regenerative components KPV-mediated inflammation control: Reduced inflammatory cell infiltration (leukocytes, mast cells) Enhanced re-epithelialization and granulation tissue formation Improved collagen deposition and organization in healing wounds Smaller scar formation demonstrated in KPV studies, complementing tissue repair components Antimicrobial effects from KPV against S
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