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Furthermore, we provide first data that the GSH level in human neurons may be mediated by NMDAR activation since the protective effect of enhanced network activity could be blocked with an NMDAR antagonist APV, consistent with the previous report (Fig

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181,182 In the apoptosis pathway, Fas ligation changes its conformational structure, allowing signaling molecules (i.e., FADD, cFLIP, and procaspase-8) to bind to the intracellular C-terminal signaling death domain of the receptor