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In the context of therapeutic response and resistance, accumulating evidence indicates that ROS contribute to the maintenance and reinforcement of resistance to platinum-based chemotherapy through adaptive mechanisms such as activation of the DNA damage response (ATR-Chk1), remodeling of ROS-regulated phosphorylation networks (e.g., CAMK2G-ITPKB), alterations in mitochondrial dynamics, and loss of MAPK regulators such as MKP3

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Notably, EV-associated DAMPs have been found in stored red blood concentrates [238], suggesting that eryptosis-mediated generation of EVs might exert immunomodulatory DAMP-triggered effects

The application of nitric oxide for ocular hypertension treatment

The ferroptosis activator RSL-3 and the PI3K/Akt pathway inhibitor LY294002 reversed Neuritins inhibitory effect on ferroptosis, indicating that Neuritin suppresses neuronal ferroptosis via PI3K/Akt pathway activation (Song et al., 2025)

Tramutola A, Arena A, Cini C, Butterfield DA, Barone E