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It was designed to be a therapeutic agent for neurodegenerative diseases due to its potential to facilitate synaptogenesis, the formation of new synapses between neurons

Taken together, if a primary autoimmune hypothesis of MS pathogenesis is correct, it is surprising that potent anti-inflammation would not be more beneficial for the progressive phase

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[13] [18] Anti-Inflammatory and Neuroprotective Effects Reduces pro-inflammatory cytokines (TNF-, IL-6) and promotes M1-to-M2 macrophage shift