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Conclusion: FOXO4-DRI promotes the nuclear export of phosphorylated P53 by inhibiting the binding of FOXO4 to P53 in endothelial cells, thereby facilitating the apoptosis of senescent endothelial cells and alleviating aging

This section consolidates the major molecular mechanisms through which luteolin exerts therapeutic effects, highlighting cross-talk among pathways to illustrate its systems-level regulatory profile

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Roles of CircRNAs in Autoimmune Diseases

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