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These factors contribute to tissue destruction, overstimulation of pain nerve fibers, and, in the case of endometriosis, further enhance the synthesis of "abnormal" estrogens by endometrial implants
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This is a crucial interaction that should be investigated using GLP-1 RAs in women
it is frequently suggested for long-term deficiency cases
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Nat Microbiol (2017) 2:17121
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