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Expression of glyoxalase, glutathione peroxidase and glutathione S-transferase isoenzymes in different bovine tissues
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Each of these molecules plays a specific role in orchestrating the repair process
Pharmacological targeting of CHAC1-mediated processes therefore represents a rational therapeutic strategy, potentially disrupting the cycle of GSH depletion, ER stress amplification and iron-dependent peroxidation that drives neuronal loss
HowickJKoletsiDIoannidisJPAMadiganCPandisNLoefMet al
Our finding that several diseases show lowered blood NAD + levels and overall imbalanced redox metabolites is intriguing, as it suggests that a variety of disease processes increase NAD + usage, potentially by activated repair processes including PARP enzymes a major user of NAD +8 .The results have high potential relevance for clinical and research applications, follow-up of metabolic interventions and detection of disease-induced NAD + or glutathione deficiency to select patients for treatment
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