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Chan SL, Liu D, Kyriazis GA et al (2006) Mitochondrial uncoupling protein-4 regulates calcium homeostasis and sensitivity to store depletion-induced apoptosis in neural cells

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Introduction Although nitric oxide (NO) has been posited as a deleterious molecule from a physiological perspective (Naseem, 2005), respiratory diseases (Soodaeva et al., 2020), neurodegenerative diseases (Snyder, 1992), and metabolic dysfunction ( In the context of exercise, nitrate supplementation has gained interest as an ergogenic aid to enhance endurance performance ( Therefore, the purpose of this review is to present the general knowledge of the underlying physiological mechanisms affected by NO and the subsequent effect of nitrate supplementation in exercise

The molecule was designed by introducing amino-acid substitutions into the native GLP-1 backbone and attaching a fatty-acid (C18 diacid) side chain via a linker, modifications described in the peptide-engineering literature as means of resisting enzymatic degradation and promoting reversible binding to serum albumin

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