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Thus, damage to mitochondrial DNA and loss of can further increase ROS production, thereby creating a cycle of dysfunction that includes increased ROS production by the respiratory chain, activation of signal transduction pathways, and potentially, leading to apoptosis or necrosis, as well as the release of cytochrome c, which triggers apoptosis

PMC 1171222

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The reason for the increase in [bCys] under mild oxidative stress is discussed in Section E

BPC-157 accelerates that process