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Neuroprotective approaches in treating TBI are designed to address distinct mechanisms within the intricate cascade of secondary injuries
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This suggests that there might be different lipid metabolic pathways in the two subtypes of HF, and inhibiting myocardial energy deficiency, by restoring ATP-dependent LV relaxation and contraction, might cause beneficial effects in HFpEF [7]
Control mice were treated with the same amount of DMSO after diluted in PBS
Was denied for mild sleep apnea
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