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Rather than replacing lifestyle interventions, GLP-1 RAs may enhance their impactby increasing satiety, reducing compulsive urges, and possibly improving cognitive control

GSH-independent thioredoxin pathway), which may prevent GSH depletion-dependent oxidative stress and cell death 18

These findings highlight a shift from a baseline inflammatory profile associated with ocular surface damage toward a post-treatment milieu in which cytokine expression parallels improvements in tear production and PROMs

It reflects a broader shift toward treating obesity with the same level of care and understanding as other chronic diseases

GLP-1

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