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Mechanistically, it binds and activates the GHSR-1a receptor the same receptor targeted by the endogenous peptide ghrelin
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Importantly, metabolomic data revealed concurrent reductions in collagen biosynthesis intermediates (trans-3-hydroxy-L-proline, glycine, L-proline) and fibroblast-activating lysophospholipids, supporting that Retatrutide induces coordinated suppression of pro-fibrotic programs at both transcriptional and metabolic levels
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