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Mechanisms underlying inflammation in neurodegeneration

Subject of Research : Glutathione transport regulation and redox homeostasis in the endoplasmic reticulum Article Title : SLC33A1 exports oxidized glutathione to maintain endoplasmic reticulum redox homeostasis News Publication Date : April 17, 2026 Web References : 10.1038/s41556-026-01922-y Image Credits : Lori Chertoff / The Rockefeller University Keywords : Antioxidants, Endoplasmic reticulum, Protein folding, Redox homeostasis, Glutathione transport, SLC33A1, Neurodegeneration, Cancer, Mitochondria, Cellular metabolism, Molecular transporters Tags: cancer and protein foldingcellular redox regulationdisulfide bond formation in proteinsendoplasmic reticulum redox homeostasisER protein maturation and traffickingglutathione role in protein foldingliposome-based assay for protein studyneurodegenerative disorder mechanismsoxidative environment in ERoxidized glutathione (GSSG) transportprotein misfolding diseasesSLC33A1 transporter function

Nature Communications , 9 (1), 2341 Scirgolea, C., Sottile, R., De Luca, M., Susana, A., Carnevale, S., Puccio, S., Ferrari, V., Lise, V., Contarini, G., Scarpa, A., Scamardella, E., Feno, S., Camisaschi, C., De Simone, G., Basso, G., Giuliano, D., Mazza, E

Foley, R

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Vascular recruitment the organised attraction of new blood vessel ingrowth into healing tissue is considered a central mechanism across BPC-157s cross-tissue cytoprotective effects and is described as a defining feature distinguishing it from other growth factors in the healing biology literature