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Three pentacyclic triterpenes protect H9c2 cardiomyoblast cells against high-glucose-induced injury

With GSH depleted, TCR signal transduction is blunted, reducing T-cell proliferation and protective cytokine output

Taken together, our findings support a model in which GCLC down-regulation leads to impaired GSH biosynthesis, disrupted redox balance, and increased oxidative damagefactors that converge to promote dopaminergic neuron vulnerability

Preclinical timelines run over weeks meaningful model-level changes appear across roughly weeks 412, not in the first days