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The prolonged expression of flavonoid biosynthetic genes reported in several stress-tolerant genotypes (Yadav et al., 2021) is consistent with this hypothesis, although direct mechanistic links between stress memory and flavonoid-specific chromatin regulation remain to be established
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Reported dosages are specific to the animal model and are not intended to represent human use
As the primary factors for the reduced anticancer effect 1) the reduced delivery and uptake of the anticancer agent, 2) the need for the conversion of molecular oxygen (O 2 ) into reactive oxygen species (ROS), 3) the preferred therapeutic effect against highly proliferating cells, which is reduced under hypoxic conditions, or 4) adaptation of the cancer cells by promoting cell survival and upregulating certain resistance pathways, has been suggested 1,2,3,4,5
S., Masternak, M
