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Direct SlC7A11 overexpression in cardiac myocytes boosted GSH levels and reduced cardiac ferroptosis and injury, suggesting that it is feasible to fortify GSH levels by increasing SLC7A11 [76]

Future research should focus on further elucidating the multidimensional mechanisms of the carnitine pool in the regulation of intercellular communication and metabolic processes and exploring its important role in maintaining the physiological balance and coping with pathological conditions

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