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Taken together, the above-mentioned data suggest that GSH might serve as an important wound signal that triggers wound-induced systemic [Ca 2+ ] cyt transmission, activates JA biosynthesis, and regulates plant defense responses
Targeting interleukin 4 receptor alpha on tumor-associated macrophages reduces the pro-tumor macrophage phenotype
Unfortunately, most of the time, they will not get better either
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However, significant limitations remain regarding data availability, model generalizability, and the translation from computational predictions to experimental validation
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