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Fortunately, we were already positioned well before GLP-1s became a mainstream topic, says Dritsas
** The result depends on individual metabolism
The triple agonist mechanism targets GLP-1, GIP, and glucagon receptors simultaneously, creating appetite suppression and metabolic enhancement that single-receptor medications cannot match
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Clinical translation requires human CSF pharmacokinetic assays to prove BBB bypass, a rigorous dose-escalation Phase I trial to establish a safe human NOAEL for chronic dosing, and an RCT utilizing precise neurocognitive testing (e.g., MoCA) to confirm that murine transcriptomic shifts yield functional neuroprotection in humans
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