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Relapse is driven by Tumor Initiating Cells (TICs), a subset of tumor cells capable of regrowing the tumor and highly resistant to therapy

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In murine intestinal inflammation models it has been shown to downshift NF-B and MAPK signaling alongside related cytokine markers

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26, 34, 44, 45 After BDL, the biliary epithelium proliferates and cholangiocytes secrete abundant amounts of these neuroendocrine factors to support the increased demands of the proliferating biliary tree