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In this study, GSH inhibited ferroptosis by activating the SLC7A11/GPX4 axis and reducing LPO levels to ultimately alleviate graft injury and promote survival
In this signalling the IGF-1 receptor (IGF-1R) is unquestionable, the predominating single factor
In septic lung injury, elevated lactate levels exacerbate alveolar epithelial cell ferroptosis through lactylated histone H3 lysine 18 (H3K18la)-mediated upregulation of METTL3, which enhances m6A modification of ACSL4, ultimately contributing to the development of acute respiratory distress syndrome (82)
Diets Physiological fuel values were calculated from modified Atwater factors (3.5 kcal/g carbohydrate
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