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The stacking of appetite-suppressing compounds increases the risk of inadvertent undereating
Our findings demonstrated that the mouse intestinal pH was neutral to weakly alkaline from the duodenum to the rectum, and the amount of hydrogen contained in the large intestine was increased by administration of the Si-based agent (Fig
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The mechanism runs through three interconnected pathways: glycemic transition (rapid reduction in blood glucose triggers compensatory vascular changes), fluid redistribution (GLP-1 receptor activation in the kidneys alters sodium reabsorption, causing transient intravascular volume contraction), and gastric mechanism overlap (nausea-induced dehydration compounds the electrolyte imbalance)
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