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Moderate mechanical stress could activate Nrf2/GPX4/HO-1 axis and inhibit NF-B pathway, therefore reducing chondrocyte ferroptosis [109]

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This raises the problem of the source of NADPH in erythrocytes

Owing to severe injury or congenital deformity, osteogenesis insufficiency, rheumatoid arthritis or osteoporosis, the long and limited self-healing process of bone grafts cannot fully meet the requirements of timely bone regeneration sometimes [3-5]

The mechanistic target of rapamycin complex 1 (mTORC1) is a kinase which regulates anabolic metabolism, cell growth and proliferation 7,8