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Given the experimental evidence mentioned above, investigating new proeryptotic and antieryptotic molecules is essential for expanding our understanding of the cellular mechanisms underlying the programmed death of erythrocytes and for opening new avenues in the study of pathologies associated with eryptosis

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For example, the researchers conducted experiments with cartilage cells and noted that FOXO4-DRI [may] remove the senescent cells in PDL9 chondrocytes

BMC Endocr Disord 24(1):177