It ameliorates various forms of experimental liver injury: attenuating drug-induced hepatic lipid peroxidation and glutathione depletion, improving D-galactosamine-induced transaminase levels and hepatocyte necrosis, hemorrhage, inflammatory infiltration, and reducing fibrosis, inflammation, lipid peroxidation, intestinal permeability, and systemic LPS levels in carbon tetrachloride-induced models 106,158,159
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