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Vleminckx S, Virgilio N, Asserin J, Prawitt J, Silva CIF
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The characterization of transporters like SLC33A1 elucidates not only fundamental biochemical pathways but also reveals new classes of druggable proteins involved in disease pathogenesis
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GSH is the thiol source for gliotoxin, which is essential for its function, and deletion of gliG results in the complete abrogation of gliotoxin biosynthesis (Figure 3)
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