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Mouse models allow elegant mechanistic dissection, but the complexity of human MASHespecially in the context of metabolic heterogeneitydemands cautious interpretation
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This oxidative damage activates signaling pathways that promote inflammation and fibrosis, leading to glomerular hypertrophy, mesangial expansion, and tubulointerstitial fibrosis [183]
*Redox Biology*, 10, 274-284
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