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The functional importance of the ASPL-VCP interaction in mediating VCP phosphorylation by ULK is further supported by our observation that the SG disassembly defects in cells expressing disease-causing VCP mutants or VCP bindingdefective ASPL mutants, both of which disrupt the ASPL-VCP interaction, were rescued by introducing phosphomimetic residues to bypass ULK-mediated phosphorylation
Moreover, the enhanced mental clarity and focus resulting from adequate vitamin levels can support your personal and professional growth
[A: 12:45, V: 12:25]
Gago-Dominguez et al
T cells in osteoarthritis: alterations and beyond
