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In conclusion, BPC 157 promotes the ex vivo outgrowth of tendon fibroblasts from tendon explants, cell survival under stress, and the in vitro migration of tendon fibroblasts, which is likely mediated by the activation of the FAK-paxillin pathway
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Our findings demonstrated that GSH inhibition leads to synthetic lethality in VRK2-deficient PC cells, which exhibits impaired expression of SLC7A11 at PM level
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PubMed Central , Journal of Hepatology , vol
Both in vitro and in vivo research involving rat tendons has shown that BPC-157 promotes collateralization and boosts fibroblast density in the setting of tendon, ligament, and bone injury
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