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This may be due to the accumulation or loss of other intermediates in the EGT synthetic pathway, for example, possibly an accumulation of -glutamyl cysteine in the egtD mutant, which has differing redox buffering capacities
Cell migration and repair coordination TB-4s role in actin sequestration and cytoskeletal flexibility is critical for cell migration, a key step in wound closure and regeneration
K., DeGrado, J
Heterotypic interactions enabled by polarized neutrophil microdomains mediate thromboinflammatory injury
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