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The EC 50 for TGR5 ligand stimulated cAMP formation and PI hydrolysis is close to the luminal bile acids concentrations and suggests that under physiological conditions it is likely that L cells would be activated by postprandial bile acids and induce GLP-1 and PYY release

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With good controllability and biocompatibility, they permit stage specific, dynamic intervention during cartilage repair and are central to precision, adjustable strategies for osteoarthritis (OA) therapy (Table 6)

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Front Pharmacol (2021) 12:750380