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It appears that BPC 157 promotes proliferation, migration, and tube formation of human umbilical vein endothelial cells through activation of ERK1/2 phosphorylation
When GSSG accumulates excessively due to dysfunctional SLC33A1 activity, this inhibits enzymes required for proper disulfide bond formation and quality control, resulting in protein misfolding and aggregation within the ER
2001 [8] Lenton KJ, San AT, Therriault H, Cantin AM, Payette H, Wagner JR
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L.et al (2004b)
platensis extract, PCB, or chlorophyllin (data not shown) suggesting that decreased leakage of ROS was due to direct antioxidant properties of S
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