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While traditional models often address hyperandrogenism and IR as primary, parallel drivers of PCOS pathology, they frequently fail to account for the rising evidence of systemic low-grade inflammation and immune dysregulation in the syndrome, particularly the pervasive role of the gut-metabolic axis [2, 16, 17]
Access to these channels significantly expands the addressable patient population and supports meaningful volume growth
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Is modulation of oxidative stress an answer
[10] [11] Symptoms warranting thyroid function assessment include unexplained fatigue, weight changes beyond those expected from GLP-1 therapy, changes in heart rate, mood alterations, temperature intolerance, or changes in bowel habits
Source: FG Newswire
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