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10.3389/fnins.2023.1209527 5 BarnesP

More specifically, the researchers posited that the peptide may have enhanced the expression of Ki-67 and CD98hc that are less intensively synthesized during cell aging. Epithalon also seemed to inhibit the activity of Caspase-3, a crucial enzyme believed to facilitate apoptosis (programmed cell death)

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doi: 10.1007/s11064-017-2250-8 233 ShiLZhangZLiLHolscherC

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