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Fc fusion to glucagon-like peptide-1 inhibits degradation by human DPP-IV, increasing its half-life in serum and inducing a potent activity for human GLP-1 receptor activation

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Kristen R

doi: 10.3389/fendo.2022.863184 Received 26 January 2022 Accepted 01 March 2022 Published 24 March 2022 Volume 13 - 2022 Edited by Indraneel (Indi) Banerjee, The University of Manchester, United Kingdom Reviewed by Finbarr P

Forbes SC, et al

Of note, a growing number of studies support mitochondrial metabolism of LAC as a common culprit underlying psychiatric and neurodegenerative diseases such as MDD and AD as well as obesity, making it important to further understand mechanisms for the development of aberrant mitochondrial metabolism of LAC